Pakistan Journal of Pharmaceutical Sciences🔒 SSL Verified

Live Data Status: Analyzed on October 11, 2026|News & Media

Welcome to the detailed analysis for pjps.pk. This domain is officially recognized as Home | PJPS. According to their official web presence, their primary focus is: "Explore our academic journals, latest research articles, and publication resources. Discover cutting-edge pharmaceutical research and stay updated with the latest developments in the field.".

↓ Explore Articles
↓ Latest Research Articles
↓ Research Scope & Subjects
↓ Editorial Leadership
↓ Indexed & Abstracted in Leading Databases
↓ Top Alternatives to pjps.pk

Tech Stack: Next.jsReact
Link Profile: Internal Pages: 38Outbound Links: 11
Localization: en

6-Month Traffic Trend Estimate

Estimated Market Value

$1,181,250

Based on domain age, authority, and keyword relevance.

TLD Availability Risk

pjps.comTaken
pjps.netTaken
pjps.orgAvailable
pjps.ioAvailable
pjps.coAvailable
pjps.aiAvailable

Official Contact & Profiles

Socials:x.com linkedin.com facebook.com siliconplex.com

Screenshot or Official Image for pjps.pk - Home | PJPS

Visual representation extracted from pjps.pk

Content Snippets from pjps.pk

"Pakistan Journal of Pharmaceutical Sciences (PJPS) is an international, peer-reviewed, open-access journal dedicated to publishing high-quality research that advances pharmaceutical, biomedical, and medicinal sciences. Published by the Faculty of Pharmacy and Pharmaceutical Sciences, University of Karachi since 1988, PJPS provides a trusted platform for researchers, academicians, clinicians, and industry professionals worldwide to disseminate innovative discoveries, foster scientific collaboration, and accelerate the translation of research into better healthcare outcomes. We welcome original research, reviews, and emerging innovations that shape the future of pharmaceutical sciences."

"Browse the most influential research published in PJPS, ranked by citation impact."

"Abstract: Background: Dental caries are chronic infectious diseases with a high global prevalence. Its pathogenesis is closely related to the adhesion of Streptococcus mutans (S. mutans), formation of dental plaque biofilms and acid production. Currently, only a few types of anticaries drugs are available for clinical use and the long-term use of these drugs can lead to side effects, such as drug resistance and dysbiosis of the oral microbiota. Objectives: Therefore, exploring new, safe and effective anti-caries treatments is an urgent task. Screening antimicrobial drugs from herbal medicines for non-antibiotic treatment of bacteria could provide new solutions to these problems. This study aimed to screen food-borne herbal extracts with synergistic antimicrobial potential to provide a theoretical foundation for the development of natural anticaries drugs. Methods: This in-vitro study evaluated the minimum inhibitory concentration and minimum biofilm inhibitory concentration of individual plant extracts (cinnamaldehyde, eugenol and polydatin) and a 1:1 mixture of these extracts. The effects of drug combinations were assessed using the checkerboard method. The inhibitory effects of drugs against S. mutans and their antibacterial mechanisms were evaluated through scanning electron microscopy and conductivity measurements. Their ability to inhibit S. mutans biofilms was assessed through crystal violet (Beijing Solebao Technology Co.) staining and scanning electron microscopy. Their inhibition of bacterial acid production and cytotoxicity to human gingival fibroblasts were also examined. Results: The combination groups exhibited synergistic bacteriostatic effects while reducing cytotoxicity. Individual and combined plant extracts exerted bacteriostatic effects by disrupting the bacterial cell membrane, significantly inhibiting S. mutans biofilm formation while suppressing its acid production. Conclusions: The application of combined food-borne herbal extracts inhibits the growth of S. mutans and its biofilm synergistically while reducing the required drug concentration and cytotoxicity. This provides a new approach for the development of natural anticaries preparations."

Key Topics & Focus Areas for pjps.pk:

Explore Articles

Latest Research Articles

Research Scope & Subjects

Editorial Leadership

Indexed & Abstracted in Leading Databases

By comparing pjps.pk to other leading websites in its niche, marketers and researchers can identify key traffic sources and growth opportunities. Explore our related resources below to find websites similar to pjps.pk.

Frequently Asked Questions

Is pjps.pk a safe and secure website?

Yes, according to our latest analysis, we detected a valid SSL certificate ensuring a secure connection.

What is the authority score of pjps.pk?

As of October 11, 2026, pjps.pk holds an estimated domain authority score of 90/100 based on our VisitRank tracking algorithms.

What are the top alternatives to pjps.pk?

You can find the best alternatives and similar sites to pjps.pk in our explore section, which includes competitors in the News & Media sector.

Data Sources & Methodology: DNS telemetry and routing records are resolved via live root servers. Market valuation estimates are computed algorithmically based on proprietary historical indexation multiples and standardized digital asset appraisal methodologies.

Common Misspellings & Typo Domains for pjps.pk:

ppjs.pkpjs.pkpjjps.pkphps.pkpups.pk

Top Alternatives to pjps.pk

vitevino.it winds up.com situscasinopro.com aunap.gov.co bbs.com xiaobaotv.video nordeafonden.dk kunstradio.at futuregamblestudio.com zee61glasss.shop dynamicnet.ca Home

"Abstract: Background: Fibrotic liver is one of the serious pathophysiological consequences of chronic injury to the liver, may progress toward serious complications including cirrhosis and hepatic carcinoma. Objectives: Panax notoginseng is used for both therapeutic and prophylactic purposes in TCM; it is a source of Dencichine, an active substance that has been proven to exhibit antifibrotic properties in hepatic fibrosis. The present study investigated how Dencichine treatment ameliorates liver fibrosis using an animal model and a cellular fibrosis model. Methods: Randomly assigned, male C57BL/6J mice received 10% CCl4 for 15 consecutive weeks to induce hepatic fibrosis. Treatment with Dencichine was initiated from 7th week onward. To simulate in-vitro the presence of a fibrotic microenvironment, LX-2 cells were treated with PDGF-BB at a concentration of 20 ng/mL and incubated from one up to four days and then treated by various doses of Dencichine. Liver function was evaluated by measuring alanine aminotransferase (ALT), AST, HA and LN. Histopathology was evaluated by haematoxylin/eosin (HE), Masson’s trichrome and Sirius red stains. Quantitative real-time PCR and western blot analyses were used to evaluate mRNA and protein expression levels, respectively. Wound-healing assays and transwell chamber experiments were used to assess cell migration; EdU proliferation detection and flow cytometry were performed to measure cell proliferation and cell cycle, respectively. Results: The most important results are a considerable reduction of serum concentrations of AST and ALT, HA and laminin in CCl4-treated mice decreased liver injury and ECM deposition. Furthermore, this compound downregulated the expression of cyclin D1 and cyclin E1 and blocked the activation of PI3K and AKT kinases in fibrotic liver cells. Conclusion: These results indicate that Dencichine may slow the development of liver fibrosis by interfering with the PI3K-AKT signaling cascade, demonstrating significant anti-scarring properties."